Komori
Other Disclosures190AChordia Therapeutics Inc.Pharmaceuticals

Notice of Publication of a Research Paper on the Results of Our Joint Research with Kyoto University on the CDK12 Inhibitor CTX-439

Results from joint research with Kyoto University on the CDK12/13 inhibitor CTX-439 were published in the academic journal Cell Death & Disease. The study indicated that loss of CDK10 function may increase sensitivity to CTX-439.

Worth reading
49Recurring
0–100. This measures how worthwhile something is to read, not how good or bad the company is.
Direction
Improvement
Direction inferred from the company’s description.
Scale
—
Displayed only when the document provides numerical figures.
Wording
Conditional
How definitive the wording is.

Announced details

CDK12 inhibitor CTX-439CDK12/13 inhibitor CTX-439
Applicable field (uses covered by this announcement)

Breast and ovarian cancer; patient stratification and treatment strategies in cancer therapy

What the company reported numerically
Quantitative Effects

Confirmed that loss of CDK10 function enhances the antitumor effect of CTX-439

Relationship between the counterparty and the company
Chordia Therapeutics Inc.190A
Relationship with the Company

Joint research relationship concerning the identification of sensitivity biomarkers for CTX-439.

CounterpartyKyoto University

Professor Yusa Kosuke (遊佐宏介) of the Institute for Life and Medical Sciences, Kyoto University, is the principal investigator. Other information, including the location, capital, and year established, is not disclosed.

In this announcement the company addresses none of Contract Value / Offering price / Impact on Business Performance.

Source (materials cited by the company)

Cell Death & Disease、Nature Portfolio

Announced details
ItemDetails
Names of Products / Services CoveredCDK12 inhibitor CTX-439, CDK12/13 inhibitor CTX-439
CounterpartyKyoto University
Counterparty overviewProfessor Yusa Kosuke (遊佐宏介) of the Institute for Life and Medical Sciences, Kyoto University, is the principal investigator. Other information, including the location, capital, and year established, is not disclosed.
Relationship with the CompanyJoint research relationship concerning the identification of sensitivity biomarkers for CTX-439.
Applicable areaBreast and ovarian cancer; patient stratification and treatment strategies in cancer therapy
Quantitative EffectsConfirmed that loss of CDK10 function enhances the antitumor effect of CTX-439
SourceCell Death & Disease、Nature Portfolio
Items this document does not state (11)
Contract ValueOffering priceContract DateLaunch dateAward/certification nameAwarding / certifying bodyRegistration number and expiry dateLocationLocation scaleUsage historyImpact on Business Performance

These are items the company does not state in this document. We do not leave them blank, nor fill them in from other sources or by inference.

The reasons the company gives

The company's own explanation, not something inferred from the numbers.

Joint research results were obtained on the identification of sensitivity biomarkers based on the mechanism of action of CTX-439.

How to read this page

This is not the documentKomori reads the disclosure the company filed and writes this summary. Figures are as stated by the company and are not audited.
Weight is not a verdictIt measures how much this changes what the company had already told the market. It is not a view on the shares.
Corrections happenIf the company reissues this disclosure, the page is rebuilt from the corrected version and the revision number moves up.
Notice of Publication of a Research Paper on the Results of Our Joint Research with Kyoto University on the CDK12 Inhibitor CTX-439 | Chordia Therapeutics Inc. (190A) timely disclosure | Komori